In the 1980s, plague tended to occur in CBGs with poor housing conditions (e.g., old homes with incomplete plumbing) and high proportions of the population living near or below the poverty line, but this second association was confounded by presence of ecotone habitat (Table 2; Figure).
We implemented the Kulldorff spatial scan statistic (9) by using SaTScan (10) to identify clusters of CBGs with high incidence rates of plague cases per 1,000 persons for each of the time frames to quantify these changes.
For example, in the 1990s, 28 (96.6%) of 29 plague-positive CBGs experienced population growth between the 1980 and 1990 censuses, in contrast to 337 (78.4%) of 430 nonplague CBGs that experienced growth.
Although we detected changes in the socioeconomic indictors associated with the locations of plague-positive CBGs, what shifting individual behavioral factors may have accompanied these trends are unknown.
Average CBGs for the basal plus correction insulin group were quite labile, with a range of 164-242 mg/dL.
Clearly, it was not routine practice to prescribe basal or prandial insulin therapy, even when subjects' CBGs warranted additional insulin therapy.
Over 50% of the total CBGs recorded represented hyperglycemia according to ADA (2007) standards.
Disadvantages of SSI as monotherapy include the failure of the health care provider to administer insulin routinely via a SSI regimen until the patient's capillary blood glucose (CBG) reaches 200 mg/dL, and the patient may experience hyperglycemia for an extended period.
The main outcome variables were CBG results less than 60 mg/dL (hypoglycemic event), CBG greater than 180 mg/dL (hyperglycemic event), and fasting blood glucose (FBS) greater than 130 mg/dL (hyperglycemic event).
or the first CBG collected before patient meal trays arrived on the nursing units.
Also, subjects were prescribed various oral medications, including sulfonylureas, biguanides, and thiazolidinediones, during hospitalization, and these various medications may have affected the CBG results.