The Specific Carbohydrate Diet
Frequently Asked Questions
Elaine wrote this about chocolate:
Chocolate is made from coca which has a a complicated effect on our physiology. Of course, cocaine comes from the same source. We were told by Dr. Haas not to use it nor carob so I am sorry, but it may not feed bad bugs, but it still is not allowed.
At another time, Elaine wrote:
The chemistry of chocolate which is cocoa which is connected in a biochemical way to cocaine. It is addictive; perhaps it is because one of the physiological effects of cocoa is "local anathesia".
Chocolate also contains ingredients which can suppress your immune system, such as metylxantil and small amounts of caffeine.
The high arginine content of chocolate gives fuel to the reproduction of certain viruses. Arginine has been shown to be an essential requirement for the replication of certain viruses.
Why are viruses so destructive? They may cause schizophrenia and autism. See article at the bottom of this page.
According to this website,cocoa contains saccharose.
Saccharose is another name for sucrose,a disaccharide.
Cocoa also contains salsolinol, a chemical that makes people crave cocoa.
In the presence of external chocolate cues, chocolate addicts were more aroused, reported greater cravings, experienced more negative affect, and also ate more chocolate than control subjects. Self-report measures on eating attitudes and behavior, body image, and depression confirmed that a relationship exists between chocolate addiction and problem eating. Chocolate addicts showed more aberrant eating behaviors and attitudes than controls, and were also significantly more depressed.
Click here to view this article on PubMed
1: Eur Neuropsychopharmacol. 2008 Oct;18(10):712-6. Epub 2008 Aug 6.
Dopamine and serotonin levels following prenatal viral infection in mouse--implications for psychiatric disorders such as schizophrenia and autism.
Winter C, Reutiman TJ, Folsom TD, Sohr R, Wolf RJ, Juckel G, Fatemi SH.
Department of Psychiatry, Charite, Campus Mitte, Berlin, Germany.
Prenatal viral infection has been associated with neurodevelopmental disorders such as schizophrenia and autism. It has previously been demonstrated that viral infection causes deleterious effects on brain structure and function in mouse offspring following late first trimester (E9) and middle-late second trimester (E18) administration of influenza virus. Neurochemical analysis following infection on E18 using this model has revealed significantly altered levels of serotonin, 5-hydroxyindoleacetic acid, and taurine, but not dopamine. In order to monitor these different patterns of monoamine expression in exposed offspring in more detail and to see if there are changes in the dopamine system at another time point, pregnant C57BL6J mice were infected with a sublethal dose of human influenza virus or sham-infected using vehicle solution on E16. Male offspring of the infected mice were collected at P0, P14, and P56, their brains removed and cerebellum dissected and flash frozen. Dopamine and serotonin levels were then measured using HPLC-ED technique. When compared to controls, there was a significant decrease in serotonin levels in the cerebella of offspring of virally exposed mice at P14. No differences in levels of dopamine were observed in exposed and control mice, although there was a significant decrease in dopamine at P14 and P56 when compared to P0. The present study shows that the serotonergic system is disrupted following prenatal viral infection, potentially modelling disruptions that occur in patients with schizophrenia and autism.
PMID: 18693086 [PubMed - indexed for MEDLINE
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